| (V1132) |
Mitochondrial thioredoxin reductase is essential for postischemic myocardial protection in mice via postischemic ROS-scavanging |
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J. C. Horstkotte, M. Schneider, P. Lange, M. Schröder, S. Schulz, R. Hinkel, C. Kiermayer, T. Ziegler, M. Brielmeier, H. Zischka, M. Conrad, C. Kupatt (München, Neuherberg) |
| (V1133) |
STAT3 controls mitochondrial NAD(P)H redox-state and reactive oxygen species production in response to β-adrenergic stimulation |
|
J. A. Saar, M. Kohlhaas, M. Hohl, B. Stapel, M. Hoch, A. von Hardenberg, M. Böhm, D. Hilfiker-Kleiner, C. Maack (Homburg/Saar, Hannover) |
| (V1134) |
Inhibition of cardiomyocyte mitochondrial STAT3 increases the formation of reactive oxygen species |
|
K. Böngler, G. Heusch, R. Schulz (Essen, Giessen) |
| (V1135) |
Postconditioning protects cardiomyocytes against reperfusion injury by limiting mitochondrial Ca2+ load and delaying pH recovery in early reperfusion |
|
T. Shahzad, W. Iraqi, S. A. Kasseckert, M. Micoogullari, W. Scharbrodt, M. Oertel, M. Parahuleva, H.-H. Tillmanns, K.-D. Schlüter, H. M. Piper, Y. Abdallah (Giessen, Düsseldorf) |
| (V1136) |
Keynote Lecture: State of the Art |
|
U. C. Hoppe (Köln) |